Correction: Immune cell senescence and chronic bone diseases: osteoimmune mechanisms and therapeutic perspectives - Report - MDSpire

Correction: Immune cell senescence and chronic bone diseases: osteoimmune mechanisms and therapeutic perspectives

  • By

  • Zhi Wen

  • Min Yu

  • Siren Li

  • Fan Zeng

  • Jing Li

  • Jiabin Li

  • Guiping Liang

  • Kang Wang

  • June 19, 2026

  • 0 min

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Correction: The Role of Immune Cell Senescence in Chronic Bone Disorders

Background

Immune cell senescence is a significant factor in the aging process, contributing to chronic inflammation and altered bone metabolism.

Data Highlights

No numerical or trial data is presented in the correction article.

Key Findings

  • The funding statement was corrected to reflect support from the Hunan Provincial Natural Science Foundation.
  • Previous funding sources included the Administration of Traditional Chinese Medicine of Jiangxi Province and Jiangxi Provincial Natural Science Foundation.
  • Immune cell senescence connects aging, inflammation, and bone metabolism disruption.

Clinical Implications

Accurate funding disclosures are vital for maintaining the integrity of research findings. Clinicians should be aware of the evolving understanding of immune mechanisms in chronic bone disorders to inform treatment approaches.

Conclusion

The correction of the funding statement enhances the credibility of the research on immune cell senescence and its role in chronic bone disorders.

Related Resources & Content

  1. Wen Z, Yu M, Li S, Zeng F, Li J, Li J, Liang G, Wang K, Frontiers in Immunology, 2026 -- Correction: The Role of Immune Cell Senescence in Chronic Bone Disorders
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  4. Archives of Toxicology — Evaluating In Vitro Bone Models for Assessing Bone Metabolism Alterations, Osteopathies, and Fracture Healing: Navigating the Complexities of Advanced Models
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  6. Effects of intermittent senolytic therapy on bone metabolism in postmenopausal women: a phase 2 randomized controlled trial - PMC
  7. Cellular senescence and other age-related mechanisms in skeletal diseases | Bone Research

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