Fetal bladder rupture in posterior urethral valves: a clinically relevant complication or a protective pop-off mechanism
By
Karolina Krzywiecka
Natalia Lekston
Zofia Sieroń
Hanna Kubik
Agnieszka Wiernik
Grzegorz Kudela
July 10, 2026
Fetal Bladder Rupture Associated with Posterior Urethral Valves
Background Posterior urethral valves are the leading cause of congenital bladder outlet obstruction in male infants, potentially leading to significant kidney damage and chronic kidney disease. Fetal bladder rupture is a severe complication that can arise from this condition, reflecting increased intravesical pressure during fetal development.
Data Highlights Parameter Findings Prenatal Diagnosis 33% of cases Main Prenatal Findings Fetal ascites, hydronephrosis, oligohydramnios Postnatal Management Endoscopic valve ablation, vesicostomy, ileocystoplasty Follow-up Outcomes Preserved kidney function, frequent bladder dysfunction
Key Findings Fetal bladder rupture is a rare but severe manifestation of PUV. 33% of cases of bladder rupture associated with PUV were diagnosed prenatally. Main prenatal findings include fetal ascites, hydronephrosis, and oligohydramnios. Postnatal management typically involves urgent urinary decompression and valve ablation. Follow-up reports indicate preserved kidney function but frequent bladder dysfunction.
Clinical Implications Multidisciplinary follow-up is essential for assessing kidney and bladder outcomes in patients with PUV and bladder rupture.
Conclusion Fetal bladder rupture associated with PUV presents significant clinical challenges.
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