Correction: The potent human CAR activator CITCO is a non-genotoxic hepatic tumour-promoting agent in humanised constitutive androstane receptor mice but not in wild-type animals - Top-Institutions - MDSpire

Correction: The potent human CAR activator CITCO is a non-genotoxic hepatic tumour-promoting agent in humanised constitutive androstane receptor mice but not in wild-type animals

  • By

  • C. J. Henderson

  • A. W. McLaren

  • A. K. MacLeod

  • D. Lin

  • A. R. Cameron

  • L. R. Chatham

  • J. Moggs

  • F. Inesta‑Vaquera

  • John P. Thomson

  • Richard R. Meehan

  • P. Chakravarty

  • M. Schwarz

  • C. Roland Wolf

  • June 27, 2026

  • 0 min

Share

Top Institutions in Oncology

Brief introduction explaining scope and methodology.

  • #1

    Mayo Clinic
    Mayo Clinic

    Rochester, Minnesota

    Key Differentiators

    • Hepatology
    • Gastroenterology

  • #2

    Massachusetts General Hospital
    Massachusetts General Hospital

    Boston, Massachusetts

    Key Differentiators

    • Hepatology
    • Gastroenterology

  • #3

    The University of Texas MD Anderson Cancer Center
    The University of Texas MD Anderson Cancer Center

    Houston, Texas

    Key Differentiators

    • Hepatology
    • Oncology

Original Source(s)

Correction: The potent human CAR activator CITCO is a non-genotoxic hepatic tumour-promoting agent in humanised constitutive androstane receptor mice but not in wild-type animals

  • by C. J. Henderson, A. W. McLaren, A. K. MacLeod, D. Lin, A. R. Cameron, L. R. Chatham, J. Moggs, F. Inesta‑Vaquera, John P. Thomson, Richard R. Meehan, P. Chakravarty, M. Schwarz, C. Roland Wolf

  • June 27, 2026

Related Content

Comparative effectiveness and safety of adjuvant trastuzumab plus pertuzumab versus trastuzumab emtansine in HER2-positive breast cancer with residual disease after neoadjuvant therapy: a real-world retrospective study

LI-RADS v2018 versus KLCA-NCC v2022: comparison of probability-based HCC categories

Chemical components of tattoo inks and their potential role in carcinogenesis and their side effects