Activation of ROS-driven genomic instability, mitochondrial depolarization, and p53-independent apoptotic cell death in human A431 epidermoid skin cancer cells by bioactive glass nanoparticles - Top-Institutions - MDSpire

Activation of ROS-driven genomic instability, mitochondrial depolarization, and p53-independent apoptotic cell death in human A431 epidermoid skin cancer cells by bioactive glass nanoparticles

  • By

  • Hanan R H Mohamed

  • Shahd Mosaad

  • Aya A. Osman

  • Alaa H. Elsewedy

  • Habiba M. Zaki

  • Mayada E. Borai

  • Gehan Safwat

  • April 2, 2026

  • 0 min

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Top Institutions in Dermatology

Brief introduction explaining scope and methodology.

  • #1

    MD Anderson Cancer Center
    MD Anderson Cancer Center

    Houston, TX

    Key Differentiators

    • Dermatologic Oncology
    • Nanomedicine
    • Cancer Biology

  • #2

    Memorial Sloan Kettering Cancer Center
    Memorial Sloan Kettering Cancer Center

    New York, NY

    Key Differentiators

    • Cutaneous Oncology
    • Nanotechnology
    • Molecular Oncology

  • #3

    Dana-Farber Cancer Institute
    Dana-Farber Cancer Institute

    Boston, MA

    Key Differentiators

    • Cancer Therapeutics
    • Nanomedicine
    • Molecular Dermatology

  • #4

    Stanford University School of Medicine
    Stanford University School of Medicine

    Stanford, CA

    Key Differentiators

    • Nanotechnology
    • Cancer Biology
    • Dermatology

  • #5

    Johns Hopkins University School of Medicine
    Johns Hopkins University School of Medicine

    Baltimore, MD

    Key Differentiators

    • Oncology
    • Nanomedicine
    • Dermatology

Original Source(s)

Activation of ROS-driven genomic instability, mitochondrial depolarization, and p53-independent apoptotic cell death in human A431 epidermoid skin cancer cells by bioactive glass nanoparticles

  • by Hanan R H Mohamed, Shahd Mosaad, Aya A. Osman, Alaa H. Elsewedy, Habiba M. Zaki, Mayada E. Borai, Gehan Safwat

  • April 2, 2026

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