Loss of ST8Sia6 mediated α-2,8-linked di-sialylation enhances T cell-dependent antibody responses and promotes autoimmunity in aged mice - Top-Institutions - MDSpire

Loss of ST8Sia6 mediated α-2,8-linked di-sialylation enhances T cell-dependent antibody responses and promotes autoimmunity in aged mice

  • By

  • Ronja Brüchert

  • Michael Hinzpeter-Schmidt

  • Bettina Röder

  • Mareike Krause

  • Stefanie Brey

  • Thomas H. Winkler

  • Falk F. R. Buettner

  • Martina Mühlenhoff

  • Lars Nitschke

  • July 2, 2026

  • 0 min

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Top Institutions in Rheumatology

Brief introduction explaining scope and methodology.

  • #1

    Johns Hopkins University and Johns Hopkins Medicine
    Johns Hopkins University and Johns Hopkins Medicine

    Baltimore, Maryland

    Key Differentiators

    • Immunology

  • #2

    Stanford University School of Medicine
    Stanford University School of Medicine

    Stanford, California

    Key Differentiators

    • Immunology

  • #3

    Massachusetts General Hospital
    Massachusetts General Hospital

    Boston, Massachusetts

    Key Differentiators

    • Immunology

  • #4

    Veterans Affairs Biorepository Brain Bank
    Veterans Affairs Biorepository Brain Bank

    Washington, D.C.

    Key Differentiators

    • Immunology

  • #5

    Banner Health
    Banner Health

    Phoenix, Arizona

    Key Differentiators

    • Immunology

Original Source(s)

Loss of ST8Sia6 mediated α-2,8-linked di-sialylation enhances T cell-dependent antibody responses and promotes autoimmunity in aged mice

  • by Ronja Brüchert, Michael Hinzpeter-Schmidt, Bettina Röder, Mareike Krause, Stefanie Brey, Thomas H. Winkler, Falk F. R. Buettner, Martina Mühlenhoff, Lars Nitschke

  • July 2, 2026

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