Biochanin A attenuates doxorubicin-induced cardiotoxicity in rats with associated modulation of PI3K/Akt/mTOR and p38 MAPK signaling - Top-Institutions - MDSpire

Biochanin A attenuates doxorubicin-induced cardiotoxicity in rats with associated modulation of PI3K/Akt/mTOR and p38 MAPK signaling

  • By

  • Mashael M. AlMutairi

  • Huda M. AlKreathy

  • Rasheed A. Shaik

  • Amani E. Alharbi

  • Rania Magadmi

  • July 7, 2026

  • 0 min

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Top Institutions in Cardiology

Brief introduction explaining scope and methodology.

  • #1

    NYU Langone Hospitals
    NYU Langone Hospitals

    New York, NY

    Key Differentiators

    • Cardiology
    • Oncology

  • #2

    The Mount Sinai Hospital
    The Mount Sinai Hospital

    New York, NY

    Key Differentiators

    • Cardiology
    • Oncology

  • #3

    Cleveland Clinic
    Cleveland Clinic

    Cleveland, OH

    Key Differentiators

    • Cardiology
    • Oncology

  • #4

    Mayo Clinic
    Mayo Clinic

    Rochester, MN

    Key Differentiators

    • Cardiology
    • Oncology

  • #5

    Johns Hopkins Hospital
    Johns Hopkins Hospital

    Baltimore, MD

    Key Differentiators

    • Cardiology
    • Oncology

Original Source(s)

Biochanin A attenuates doxorubicin-induced cardiotoxicity in rats with associated modulation of PI3K/Akt/mTOR and p38 MAPK signaling

  • by Mashael M. AlMutairi, Huda M. AlKreathy, Rasheed A. Shaik, Amani E. Alharbi, Rania Magadmi

  • July 7, 2026

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